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Our experiments in mouse models of mild hyperhomocysteinemia caused by a deficiency in cystathionine beta-synthase in CBS +/− (heterozygous deficient) mice found that increased plasma total homocysteine caused abnormal responses to endothelium-dependent vasodilators

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At last the work points to the possibility of the interchange among the different areas of the medical-scientific community, in na approach that proposes to rescue the medicine singleness and to contemplate the human health globally