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Thus, it is likely that with BPC 157 therapy, no extraosseous bone islet means the counteraction of the endochondral bone formation in extraskeletal tissues, the counteraction of vascular endothelial cells differentiation into skeletal cells, local inflammatory signals, and the release of cytokines (i.e., bone morphogenetic protein-2 and bone morphogenetic protein-4 and transforming growth factor-beta) [68]

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It does not directly stimulate fibroblast proliferation under normal culture conditions, but substantially increases fibroblast survival under hydrogen peroxide-induced oxidative stress — acting as a cytoprotective enhancer of the endogenous repair response rather than a direct mitogenic agent